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10.1007/s12031-015-0526-5 J

(2024) 27:e15297
P., Nolde, M

Further, alcohol-induced oxidative stress in the liver was also regulated by protein modifications such as protein S-glutathionylation

In terms of iron metabolism and homeostasis in immune cells, a study revealed that iron overload promotes abnormal differentiation of pathogenic T cells, including follicular helper T cells, by regulating DNA demethylation, thus exacerbating autoantibody production and the development of systemic lupus erythematosus (SLE) [26]
