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Prati D, Taioli E, Zanella A, et al
Previously, this loss of activity had been determined to be due to a RhoA GTPase-dependent internalization which could be blocked by dominant-negative RhoA constructs and by genetic introduction of the exotoxin C3, a clostridial exotoxin that inactivates RhoA by ADP-ribosylation [1]
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High levels of dietary methionine improves sitagliptin-induced hepatotoxicity by attenuating oxidative stress in hypercholesterolemic rats
doi: 10.1016/S0749-3797(03)00113-2
