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This work established the rationale for the dual modification approach [1]

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In the skin, IGF1R signaling regulates DNA repair through downstream targets PI3K/Akt (Kemp et al., 2017a, b) or through the ATR-Chk1 kinase pathway (Strozyk and Kulms, 2013)

Non-randomised studies primarily focused on assessing oxidative stress indirectly using biochemical markers and RCTs examined clinical outcomes and safety

Others found that the effect of zinc on apoptosis via caspase activity depends on the dosage: treatment of Burkitt Lymphoma cells with up to 50 M of zinc inhibited caspase 3 activation and apoptosis, whereas higher concentrations up to 100 M rather induced programmed cell death via caspase 3 [218]