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utilized a human vaginal keratinocyte cell model to reveal that internalized MNPs impaired the expression of junctional and adherence proteins, disturbed the organization of the actin cortex and influenced genes involved in oxidative stress signaling pathways and miRNAs crucial for vaginal epithelial barrier function [38]
The plot displayed the absolute difference between the two methods on the x-axis and their average on the y-axis
Glucagon-Like Peptide-1 Protects Beta-Cells Against Apoptosis by Increasing the Activity of an IGF-2/IGF-1 Receptor Autocrine Loop
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