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GSK-3 is a key mediator of PI3K-AKT-Nrf2 signaling pathway, and it can be suppressed by AKT-mediated phosphorylation (Salazar et al., 2006)
In Ni-hyperaccumulator, T
10.1038/nrm.2016.25 116 JonesD
In addition, their metabolic activity can have important consequences for the cellular redox status, and impacts the inflammatory response as well as the balance of inflammatory mediators, which can modulate epigenetic factors and cooperate or interfere with drug-adduct formation
