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10.1093/nar/gkn180 26 DitkowskiB.TrocP.GindaK.DonczewM.ChaterK
Understanding S-glutathionylation is crucial for elucidating cellular responses to redox changes and stress conditions
The inflammatory response in RA is mediated by CD4+ T cells, particularly Th1 and Th17 lymphocytes, as well as an imbalance between Th17 cells and Tregs
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Mechanisms of Jak/STAT signaling in immunity and disease
