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Other studies emphasize the crucial role of the p21 protein in maintaining the senescence phenotype [50]

For example: Severe oxidation of albumin impairs the ability of albumin to bind bilirubin, possibly due to the high affinity of bilirubin to HMA (114)

CRP binding to oxLDL and apoptotic cells occurs through phosphorylcholine, and binding to this ligand starts phagocytosis (100, 170, 175181)

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Although IGF-IR promotes EMT, it does not necessarily mean that inhibition of IGF-IR is able to reverse this phenotype, due to the compensation by other RTKs