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Selenoproteins influence the integrity of the intestinal barrier, and their reduced expression promotes increased intestinal permeability and bacterial translocation [57], which is essential in the development of inflammatory bowel diseases, metabolic disorders, and autoimmune diseases

For patients with COQ2 or COQ6 mutations, supplementation with 4-hydroxybenzoic acid (4-HBA) may enhance endogenous CoQ10 synthesis (136, 137)

For instance, the M83 mouse expressing mutant human syn demonstrates that abnormal syn accumulation is sufficient to drive neurodegenerative phenotypes including neuronal hyperactivity and vascular patterning defects, even in the absence of overt systemic inflammation [164, 165]

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