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10.1007/s12264-018-0269-8 [DOI] [PMC free article] [PubMed] [Google Scholar] Whittle B

Moreover, p53 can indirectly elevate intracellular GSH levels by regulating p21, boosting GPX4 synthesis, and subsequently inhibiting ferroptosis [149]

Attenuating ER stress with exogenous H 2 S represents a promising therapeutic strategy for PH with high translational potential ( Mesenchymal stem cells (MSCs) derived from the placenta, bone marrow, adipose, and other tissues have emerged as a promising regenerative therapy for PH over the last decade ( In vivo , CSE overexpression improved the survival of erastin-treated MSCs delivered to mice with HPH ( In vitro , CSE overexpression enhanced H 2 S production and inhibited ferroptosis-related markers in erastin-treated MSCs ( 2 S pathway induced Keap1 S-sulfhydration, which contributed to the inhibition of ferroptosis ( 2 S pathway in CMSCs inhibits ferroptosis and enhances their suppressive effect on vascular remodeling in mice with HPH ( 2 S pathway against ferroptosis in CMSCs is mediated via S-sulfhydration of Keap1 and subsequent activation of Nrf2 signaling

So less than half from the age of 25 to 60, our GHK levels fall in half on average per this study

Nature 522 , 349353 (2015)