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AML progression is sustained by leukaemia-propagating cells with stem-like characteristics (AML-LSCs), which need to be eliminated to achieve clinical remission and are the cause of relapse

Future experimental efforts can be directed at more precise characterization of the underlying mechanism of action of the drug and elucidating other potential metabolic targets of halofantrine in L

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Zn can prevent apoptosis and thus prevent oxidative damage in the brain (Kocatrk et al., 1996)

Shibata F, Miyama K, Shinoda F, Mizumoto J, Takano K, Nakagawa H (2004) Fibroblast growth-stimulating activity of S100A9 (MRP-14)