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Clinical trials have shown that the role of COX-2 in human carcinogenesis is attributed to its overexpression in prostate cancer tissues (Rafailov et al., 2007)

10.1016/j.cub.2018.05.094 198 StehlingO.WilbrechtC.LillR

This browning is mediated through multiple pathways including SIRT1/PGC-1 activation, increased expression of thermogenic genes (UCP1, PRDM16, PPAR), and enhanced mitochondrial biogenesis

The additive effects observed when combining NNMT inhibition with exercise indicate that the compound influences molecular pathways distinct from those activated by physical training alone

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