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In: Cluzan RV, Pecking AP, Lokiec FM, eds
Polyak K, Weinberg RA
This is because oxidative damage can result in genomic instability, DNA mutations, and tumorigenesis and progression
Similarly, in a xenograft mouse model where MDA-MB-231 breast cancer cells were co-injected with wild-type or Cav-1-deficient fibroblasts, the Cav-1-deficient fibroblasts led to enhanced glycolytic enzyme synthesis to provide energy-rich metabolites (e.g., lactate), thus increasing cancer growth and angiogenesis [156]
M.EikelenboomP.et al
