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Program in Sciences and Innovation in Medicine at Universidad del Desarrollo Inhibition of SLC11A1-Mediated Lysosomal Iron Accumulation in Microglia Promotes Repair Following White Matter Stroke This study in Advaced Science elucidates a critical pathogenic mechanism in White Matter Stroke (WMS) recovery, identifying the H+/Fe2+ antiporter SLC11A1 as a primary driver of microglial dysfunction via time-dependent (12h14d) lysosomal iron accumulation

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