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Whereas the literature typically reports the role of GSH as an antioxidant, the results of the present work appear to tentatively show that GSH modulation is a consequence of sustained cognitive effort (previous section) and that it is associated with the quality of cognitive performance, at least in young adults (present section)

Researchers became interested when preclinical studies demonstrated that adding glucagon receptor agonism to incretin-based therapy produced metabolic effects that exceeded those of GLP-1 or GIP activation aloneparticularly in energy expenditure and hepatic lipid metabolism

doi: 10.3389/fgene.2014.00201

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