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This is not surprising, given that meta-analytic pooling of ACC GSH studies in schizophrenia reveal a small overall effect size [24], suggesting the possibility of heterogeneity in the GSH levels and thus redox status among patients

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In the concanavalin A (ConA)-induced autoimmune hepatitis model, pretreatment with RvD1 or RvE1 markedly reduced the release of TNF-, IFN-, IL-2, IL-1, and IL-6, while simultaneously suppressing CD4 and CD8 T-cell infiltration through the inhibition of NF-B and AP-1 signaling [244]

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