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Under neuropathological conditions, mitochondrial defects often lead to electron transfer processes and reduced NADPH oxidase activity, resulting in increased reactive oxygen species production, which weakens the redox buffering capacity of the cell and may damage key enzymes involved in energy metabolism (Tang, 2020)

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Read more by Darcy Jimenez Darcy became managing editor at C+D in April 2026, after joining as a senior reporter the previous August

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