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The copper concentration used in experiments critically influences cellular responses: low levels (0.10.1.2mmol/l) induce physiological/adaptive transcriptional changes, while higher levels (0.4-0.4.6mmol/l) trigger toxic, stress-related responses [24]
(A) The percentage of cells in sub-G1 phase
found that the IKK S-glutathionylation by glutathione disulfide amplified TNF- mediated hepatotoxicity, revealing that IKK S -glutathionylation was the potential mechanism of NAFLD [57]
A., Ashour, N
