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20 BoyceR.GlasgowS

The introduction of miR-351-5p (the target of circPtpn14) was found to reverse the 5-LOX upregulation and ER stress signaling activation caused by circPtpn14 overexpression alone and to rescue the decreased cell viability, inhibition of GPX activity, and increased blood-brain barrier permeability in vitro caused by circPtpn14 Wu et al

Obesity is associated with macrophage accumulation in adipose tissue

To eat or not to eat mitochondria

Cells treated with Triton X-100 were used as a positive control and to induce the maximum fluorescence intensity
