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eLife, 14, e12270663

Onderzoek naar Cellulaire Regeneratie en Homeostase

Cha and co-authors showed that Grx3 has a role in cardiomyocyte regulates contractility and cardiac hypertrophy and cardiomyocytes with heterozygous deletion of Grx3 (Grx3+/-) showed a decrease in Ca 2+ sensitivity in cardiomyocyte filaments

These findings indicate that Rtbdn's function is linked to its RF-binding abilities and leads us to hypothesize that downstream metabolic and degenerative phenotypes associated with Rtbdn elimination are due to its function as a flavin binding protein [19]